Citation: | JI Jin-long, CHEN Li-ping, HAN Yun, LIU Ying-lei, TANG Wei-chun, YOU Jun. Study on the correlation between the expression and methylation of LCHAD gene and pathological pregnancy state[J]. Journal of Bengbu Medical University, 2023, 48(7): 917-921. DOI: 10.13898/j.cnki.issn.1000-2200.2023.07.014 |
[1] |
淮静, 杨孜, 易雁鸿, 等. 普伐他汀对子痫前期样小鼠模型中长链脂肪酸氧化酶的调节作用[J]. 中华妇产科杂志, 2018, 53(3): 183.
|
[2] |
BARTHA JL, VISIEDO F, FERNÁNDEZ-DEUDERO A, et al. Decreased mitochondrial fatty acid oxidation in placentas from women with preeclampsia[J]. Placenta, 2012, 33(2): 132. doi: 10.1016/j.placenta.2011.11.027
|
[3] |
DING X, YANG Z, HAN Y, et al. Adverse factors increase preeclampsia-like changes in pregnant mice with abnormal lipid metabolism[J]. Chin Med J(Engl), 2014, 127(15): 2814.
|
[4] |
DING X, YANG Z, HAN Y, et al. Fatty acid oxidation changes and the correlation with oxidative stress in different preeclampsia-like mouse models[J]. PLoS One, 2014, 9(10): e109554. doi: 10.1371/journal.pone.0109554
|
[5] |
GUO F, YAN L, GUO H, et al. The transcriptome and DNA methylome landscapes of human primordial germ cells[J]. Cell, 2015, 161(6): 1437. doi: 10.1016/j.cell.2015.05.015
|
[6] |
HUAI J, YANG Z, YI YH, et al. Role of mammalian target of rapamycin signaling pathway in regulation of fatty acid oxidation in a preeclampsia-like mouse model treated with pravastatin[J]. Chin Med J(Engl), 2019, 132(6): 671.
|
[7] |
DING X, YANG Z, HAN Y, et al. Correlation of long-chain fatty acid oxidation with oxidative stress and inflammation in pre-eclampsia-like mouse models[J]. Placenta, 2015, 36(12): 1442. doi: 10.1016/j.placenta.2015.10.014
|
[8] |
NATARAJAN SK, IBDAH JA. Role of 3-hydroxy fatty acid-induced hepatic lipotoxicity in acute fatty liver of pregnancy[J]. Int J Mol Sci, 2018, 19(1): 322. doi: 10.3390/ijms19010322
|
[9] |
BARTHA JL, BUGATTO F, FERNÁNDEZ-DEUDERO Á, et al. Tissue specific expression of human fatty acid oxidation enzyme genes in late pregnancy[J]. Lipids Health Dis, 2016, 15(1): 200. doi: 10.1186/s12944-016-0373-6
|
[10] |
ROVELLI V, MANZONI F, VIAU K, et al. Clinical and biochemical outcome of patients with very long-chain acyl-CoA dehydrogenase deficiency[J]. Mol Genet Metab, 2019, 127(1): 64. doi: 10.1016/j.ymgme.2019.04.001
|
[11] |
SANDVOB M, POTTHAST AB, VON VERSEN-HÖYNCK F, et al. HELLP syndrome: altered hypoxic response of the fatty acid oxidation regulator SIRT 4[J]. Reprod Sci, 2017, 24(4): 568. doi: 10.1177/1933719116667216
|
[12] |
孟然, 杨孜, 王海玲, 等. 早发重度子痫前期、HELLP综合征及抗磷脂综合征对胎盘滋养细胞LCHAD基因甲基化的影响及其与基因mRNA表达相关性的研究[J]. 中华妇产科杂志, 2016, 51(4): 270. doi: 10.3760/cma.j.issn.0529-567x.2016.04.006
|
[13] |
NANDI AA, WADHWANI NS, JOSHI SR. Altered metabolic homeostasis between vitamin D and long chain polyunsaturated fatty acids in preeclampsia[J]. Med Hypotheses, 2017, 100(3): 31.
|
[14] |
YU H, YANG Z, DING X, et al. Effects of serum from patients with early-onset pre-eclampsia, HELLP syndrome, and antiphospholipid syndrome on fatty acid oxidation in trophoblast cells[J]. Arch Gynecol Obstet, 2015, 292(3): 559. doi: 10.1007/s00404-015-3669-2
|
[15] |
YANG X, XU P, ZHANG F, et al. AMPK hyper-activation alters fatty acids metabolism and impairs invasiveness of trophoblasts in preeclampsia[J]. Cell Physiol Biochem, 2018, 49(2): 578. doi: 10.1159/000492995
|
[16] |
LAROSA M, LE GUERN V, MOREL N, et al. Evaluation of the severe preeclampsia classification criterion for antiphospholipid syndrome in a study of 40 patients[J]. Arthritis Res Ther, 2021, 23(1): 134. doi: 10.1186/s13075-021-02518-7
|
[17] |
YU X, HE L. Aspirin and heparin in the treatment of recurrent spontaneous abortion associated with antiphospholipid antibody syndrome: A systematic review and meta-analysis[J]. Exp Ther Med, 2021, 21(1): 57.
|
[18] |
FUKAI T, USHIO-FUKAI M. Cross-talk between NADPH oxidase and mitochondria: role in ROS signaling and angiogenesis[J]. Cells, 2020, 9(8): 1849. doi: 10.3390/cells9081849
|